(1.5 hrs. / 0.15 CEUs)
Historically, pressure injury etiology has revolved around ischemic changes in the skin, and soft tissue. However, recent evidence has demonstrated that tissue deformation of the skin and soft tissue has earlier implications in pressure injury development versus just ischemia alone.
Currently there are various levels of clinical knowledge and experience in the healthcare community when it comes to understanding pressure injury etiology and appropriate management strategies. Unfortunately, most healthcare practitioners who are actively participating in wound management, do not fully understand the pathophysiology of pressure injuries. There is also a lack of clear understanding of the extrinsic and intrinsic risks, which need to be correctly managed for successful outcomes.
This ninety minute interactive session will review the current best evidence of pressure injury pathophysiology, with a focus on the new understanding of the significant role tissue deformation plays in the development of pressure injuries. Discussions will revolve around the differences in the development of a superficial pressure injury versus a suspected deep tissue injury. In addition, the intrinsic and extrinsic risk factors commonly associated with pressure injuries will also be discussed along with strategies to minimize the risks. This program will also review new terminology and the revised staging guidelines from the National Pressure Ulcer Advisory Panel published in April 2016.
Objectives: By the end of the presentation, participants will be able to:
- Define suspected deep tissue pressure injury according to the NPUAP staging guidelines
- Explain three differences in progression of superficial versus deep pressure injuries
- Define the new pressure injury staging guidelines according to the NPUAP staging guidelines.
- List the four accepted pathological mechanisms causing a pressure injury.
- Describe three intrinsic and the four extrinsic risk factors in developing pressure injuries.
- Contrast three differences in the pathology of pressure injuries related to deformation versus ischemia.